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Administration of alloxan to a rat for up to 1 yr results in a diabetes with a protracted course, in which after 2-3 mo the animals suffer from a moderate hyperglycemia, while after 8-9 mo of the treatment, their glucose tolerance is impaired. In this study, the free radical parameters (urinary reactive oxygen metabolites (URO(4)(-))/nucleotide ratio and thiobarbituric acid-reactive substances) and the activities of hepatic glutathione-S-transferase and glutathione reductase were determined as well as the oxidative stress markers (lipid peroxides, advanced oxidation protein product, and 4-hydroxynonenal protein adducts) were compared in alloxan-treated control rats and in long-term diabetic animals. The values of URO(4)(-)/nucleotides ratio and lipid peroxides in long-term diabetic rats were higher than those of controls, but there was no increase in oxidatively modified proteins and these parameters did not reach the values in short-term diabetic rats. By contrast, the parameters of oxidant stress in long-term diabetic rats were less significant than those in short-term diabetic rats. Antioxidants were also measured to assess the ability of the liver to deal with oxidant stress. The activities of hepatic glutathione-S-transferase and glutathione reductase were
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